Mitochondrial Disease: Difference between revisions
No edit summary |
No edit summary |
||
| Line 15: | Line 15: | ||
[https://www.pnas.org/doi/10.1073/pnas.1621511114 Hypoxia treatment reverses neurodegenerative disease in a mouse model of Leigh syndrome] | [https://www.pnas.org/doi/10.1073/pnas.1621511114 Hypoxia treatment reverses neurodegenerative disease in a mouse model of Leigh syndrome] | ||
[https://pubmed.ncbi.nlm.nih.gov/33752971/ Hypoxia ameliorates brain hyperoxia and NAD(+) deficiency in a murine model of Leigh syndrome] | |||
Jain IH, Zazzeron L, Goli R, et al. Hypoxia as a therapy for mitochondrial disease. Science. Apr 1 2016;352(6281):54-61. doi:10.1126/science.aad9642 | Jain IH, Zazzeron L, Goli R, et al. Hypoxia as a therapy for mitochondrial disease. Science. Apr 1 2016;352(6281):54-61. doi:10.1126/science.aad9642 | ||
Revision as of 17:37, 4 January 2024
This is a Stub Notice. This page has not been completed. You can work on this page by signing in and going to the Edit tab. Thanks for helping to make PedsAnesthesia.Net Wiki useful.
Go to the Main Page to see the Topic Outline.
Go to the Generalized Suggested Outline for information on case-specific details for each page.
Go to the Test Page for examples on how to use references in the page.
Relevant Article Depot:
A review of anaesthetic outcomes in patients with genetically confirmed mitochondrial disorders
Mitochondrial Disease and Anesthesia
References:
Anesthetic Hypersensitivity in a Case-Controlled Series of Patients With Mitochondrial Disease
Anesthetic considerations in patients with mitochondrial defects
Hypoxia treatment reverses neurodegenerative disease in a mouse model of Leigh syndrome
Hypoxia ameliorates brain hyperoxia and NAD(+) deficiency in a murine model of Leigh syndrome
Jain IH, Zazzeron L, Goli R, et al. Hypoxia as a therapy for mitochondrial disease. Science. Apr 1 2016;352(6281):54-61. doi:10.1126/science.aad9642
Open Anesthesia: Anesthetic Considerations in Patients with Mitochondrial Disease
Volatile anaesthetic toxicity in the genetic mitochondrial disease Leigh syndrome